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2015). These research, taken jointly, suggest that, Whilst a immediate activation of GPR55 or PPAR?? takes place, PEA can develop its anti?inflammatory action inside the intestine also by using indirect activation of CB1 and CB2 receptors, likely because of the flexibility of this compound to potentiate the action of endocannabinoids at these recep
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Clinicaltrials.gov was looked for registered review protocols. The keywords and phrases “palmitoylethanolamide” and “ache” resulted in thirteen entries, of which 3 were accomplished. None of these fulfilled our eligibility standards.All three experiments are in step with present literature on PEA’s substantial therapeutic index, that make